Lately, I've been using a nut milk as a sort of calcium supplement. I haven't reached any consensus on whether this is improving my health or working against my goals.
8oz of this guy (1 serving) provides 500 milligrams of Calcium (I usually use 4oz/d):
http://royalhawaiianorchards.com/produc ... damia-milk
My calcium intake averages around 300 - 700mg/d of Ca prior to this nut milk product -- the upper limit is usually boosted up by bone-in sardines, where some days come in a little under 300mg. My Magnesium intake is around 400mg prior to supplementing another 300mg.
I've been using it with cacao powder + vanilla + ceylon cinnamon + sliced & soaked tigernuts to make a pseudo cereal with berries.
Calcium supplements and dementia
Re: Calcium supplements and dementia
apod, here's the unanswered question - is food, like nut milk, where the calcium is added, any different than taking an actual supplement? I haven't dug into that much, so I'd like to hear if anyone has some references.
Re: Calcium supplements and dementia
I would also be curious, from a supplement stance if calcium carbonate (eg. egg shells / coral calcium / the usual Ca additive & cheap supplement form) is ideal because it has lower bioavailability and might increase serum Ca to a lesser degree (maybe particularly in the case of a meal rich in oxalates / phytates / magnesium) or if a chelate is a smarter option because of the better targeted uptake (and lower dose?)
When researching this in the past, there were a few strong proponents against Ca Carbonate (for fear of calcification), where that always seemed like the safest option to me compared to a chelate with higher absorption / serum concentrations / half lifes.
Interestingly, I've read that a lower calcium diet increases Ca absorption & calcification, which seems somewhat paradoxical. I don't buy into most of what Ray Peat is selling, but he does have some good information on this subject.
http://link.springer.com/article/10.1007%2FPL00010637
"It is extremely important to realize that calcium deposits in soft tissues become worse when the diet is low in calcium. Some people who know about the involvement of calcium in aging, stress, and degeneration suggest eating a low calcium diet, but since we all have skeletons, dietary calcium restriction cant protect our cells, and in fact, it usually intensifies the process of calcification of the soft tissues. A deficiency of either calcium or magnesium can stimulate the parathyroid glands to produce more hormone (parathyroid hormone, PTH), which increases calcium absorption, but also removes calcium from the bones. This hormone, responding to a dietary calcium or magnesium deficiency, is an important factor in causing cells to take up too much calcium, and its excess is associated with many inflammatory and degenerative diseases. Interleukin-6 (IL-6), an inflammatory cytokine which increases with aging, is commonly considered to have an important role in the multiple processes of atrophy in old age. One of the things which can increase the production of IL-6 is the parathyroid hormone (PTH), which increases the amount of calcium circulating in the blood, partly by causing it to be removed from the bones; IL-6 stimulates the process of calcium removal from bones. Prolactin, which is increased under the influence of estrogen or serotonin, causes the body to lose calcium (drawing it from the bones), and it stimulates the secretion of PTH, which compensates for the calcium loss by increasing its mobilization from bones. Prolactins action on bone is at least partly by increasing IL-6 formation; IL-6 stimulates the release of prolactin. Serotonin and IL-6 stimulate each others secretion, and PTH and serotonin each stimulate the others release. A low protein diet, similar to that eaten by a large proportion of women (0.8 g/kg of body weight) increases PTH, and so probably contributes to the development of osteoporosis and the diseases of calcification. The most important calcium paradox is that medical journals (e.g., International J. of Cardiology, Dec., 2002) are still promoting the idea that eating too much calcium causes hardening of the arteries and other diseases of calcification."
When researching this in the past, there were a few strong proponents against Ca Carbonate (for fear of calcification), where that always seemed like the safest option to me compared to a chelate with higher absorption / serum concentrations / half lifes.
Interestingly, I've read that a lower calcium diet increases Ca absorption & calcification, which seems somewhat paradoxical. I don't buy into most of what Ray Peat is selling, but he does have some good information on this subject.
http://link.springer.com/article/10.1007%2FPL00010637
"It is extremely important to realize that calcium deposits in soft tissues become worse when the diet is low in calcium. Some people who know about the involvement of calcium in aging, stress, and degeneration suggest eating a low calcium diet, but since we all have skeletons, dietary calcium restriction cant protect our cells, and in fact, it usually intensifies the process of calcification of the soft tissues. A deficiency of either calcium or magnesium can stimulate the parathyroid glands to produce more hormone (parathyroid hormone, PTH), which increases calcium absorption, but also removes calcium from the bones. This hormone, responding to a dietary calcium or magnesium deficiency, is an important factor in causing cells to take up too much calcium, and its excess is associated with many inflammatory and degenerative diseases. Interleukin-6 (IL-6), an inflammatory cytokine which increases with aging, is commonly considered to have an important role in the multiple processes of atrophy in old age. One of the things which can increase the production of IL-6 is the parathyroid hormone (PTH), which increases the amount of calcium circulating in the blood, partly by causing it to be removed from the bones; IL-6 stimulates the process of calcium removal from bones. Prolactin, which is increased under the influence of estrogen or serotonin, causes the body to lose calcium (drawing it from the bones), and it stimulates the secretion of PTH, which compensates for the calcium loss by increasing its mobilization from bones. Prolactins action on bone is at least partly by increasing IL-6 formation; IL-6 stimulates the release of prolactin. Serotonin and IL-6 stimulate each others secretion, and PTH and serotonin each stimulate the others release. A low protein diet, similar to that eaten by a large proportion of women (0.8 g/kg of body weight) increases PTH, and so probably contributes to the development of osteoporosis and the diseases of calcification. The most important calcium paradox is that medical journals (e.g., International J. of Cardiology, Dec., 2002) are still promoting the idea that eating too much calcium causes hardening of the arteries and other diseases of calcification."
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Re: Calcium supplements and dementia
"I've read that a lower calcium diet increases Ca absorption"
same with magnesium..I like the Albion stuff which is chelated, I take the Mg-glycinate (600mg) is well absorbed. Absorption depends on the gut condition and various other co factors.
With Mg if it is not in the soil even if it looks good it will not be in the food so I chose organic stuff as much as I can get.
Dairy has about 10:1 Ca : Mg not too good.
I avoid any Ca in supplements a low value is ok tho.
same with magnesium..I like the Albion stuff which is chelated, I take the Mg-glycinate (600mg) is well absorbed. Absorption depends on the gut condition and various other co factors.
With Mg if it is not in the soil even if it looks good it will not be in the food so I chose organic stuff as much as I can get.
Dairy has about 10:1 Ca : Mg not too good.
I avoid any Ca in supplements a low value is ok tho.
Re: Calcium supplements and dementia
Stavia, do we need to now chase Calcium biomarker (only saw Potassium and Sodium in my 1st labs, both normal) as it relates to AD??
MAC
E3/E4-59/MALE
E3/E4-59/MALE
Re: RE: Re: Calcium supplements and dementia
I don't know of evidence that it's critical in AD Mac.MAC wrote:Stavia, do we need to now chase Calcium biomarker (only saw Potassium and Sodium in my 1st labs, both normal) as it relates to AD??
In addition, it's very tightly regulated by complex endocrine and bone and gut factors, and hardly dependent on dietary intake except in very unusual circumstances eg renal failure which would be obvious. Or severe dietary deficiencies. It is out of range late in most diseases affecting it.
It's not something that is usually tracked serially in a well person. I don't think your doctor missed out by not ordering it. In a clinically well person it's overwhelmingly likely to be normal.
Julie might know more - but I don't know of any way to manipulate it diet wise in a well person, and not aware of any reason to do so.
My concerns have been around people taking a large dose in a tablet in one go and what possible impact that might have downstream by the sudden blood level peak before the body's mechanisms can redistribute it.
Re: Calcium supplements and dementia
More on calcium from the MESA data.
https://www.sciencedaily.com/releases/2 ... 182621.htm
"The researchers again accounted for the same demographic and lifestyle factors that could influence heart disease risk, as in the previous analysis, and found that supplement users showed a 22 percent increased likelihood of having their coronary artery calcium scores rise higher than zero over the decade, indicating development of heart disease."
https://www.sciencedaily.com/releases/2 ... 182621.htm
"The researchers again accounted for the same demographic and lifestyle factors that could influence heart disease risk, as in the previous analysis, and found that supplement users showed a 22 percent increased likelihood of having their coronary artery calcium scores rise higher than zero over the decade, indicating development of heart disease."
Re: RE: Re: Calcium supplements and dementia
Great thought. No RCT studies alas.sarahb12 wrote:I wonder how k2 would influence all of this.