Difference between revisions of "Optimize antioxidants"
(Created page with "The eighteenth intervention to reverse mild Alzheimer's in the Bredesen Protocol is: :'''Goal''': Optimize antioxidants :'''Approach''': Mixed tocopherols and tocotrieno...") |
|||
| Line 11: | Line 11: | ||
Vitamin E 400 IU (best to use mixed [http://en.wikipedia.org/wiki/Tocopherol tocopherols], optimally including [http://en.wikipedia.org/wiki/Tocotrienol tocotrienols]). | Vitamin E 400 IU (best to use mixed [http://en.wikipedia.org/wiki/Tocopherol tocopherols], optimally including [http://en.wikipedia.org/wiki/Tocotrienol tocotrienols]). | ||
Se | Alzheimer's patients with low Se ([http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4147716/ Selenium]) in their blood have been found to have more problems with cognitive function. | ||
[https://www.sciencedaily.com/releases/2016/03/160314084821.htm bluberries] | |||
NAC ([http://onlinelibrary.wiley.com/doi/10.1002/brb3.208/full N-acetylcysteine ]) blocks oxidative damage in Alzheimer's. | |||
a-lipoic acid | Blood brain barrier dysfunction accelerates the rate of degeneration in Alzheimer's by impairing the ability of the brain to concentrate ascorbate ([http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2674290/ Vitamin C]) and other nutrients with neuroprotective properties. | ||
[http://alzdiscovery.org/cognitive-vitality/report/ala a-lipoic acid] can reduce the formation of Alzheimer's disease plaques and increase the production of acetylcholine, an important signaling molecule that is impaired in Alzheimer's disease. | |||
The next intervention is [[Optimize Zn:fCu ratio]]. | The next intervention is [[Optimize Zn:fCu ratio]]. | ||
Revision as of 13:38, 6 June 2016
The eighteenth intervention to reverse mild Alzheimer's in the Bredesen Protocol is:
- Goal: Optimize antioxidants
- Approach: Mixed tocopherols and tocotrienols, Se, blueberries, NAC, ascorbate, a-lipoic acid
- Improving and Retaining Memory Function Feb 15, 2015
Vitamin E 400 IU (best to use mixed tocopherols, optimally including tocotrienols).
Alzheimer's patients with low Se (Selenium) in their blood have been found to have more problems with cognitive function.
NAC (N-acetylcysteine ) blocks oxidative damage in Alzheimer's.
Blood brain barrier dysfunction accelerates the rate of degeneration in Alzheimer's by impairing the ability of the brain to concentrate ascorbate (Vitamin C) and other nutrients with neuroprotective properties.
a-lipoic acid can reduce the formation of Alzheimer's disease plaques and increase the production of acetylcholine, an important signaling molecule that is impaired in Alzheimer's disease.
The next intervention is Optimize Zn:fCu ratio.